Congenital contractural arachnodactyly (CA) also known as ‘Fawn calf syndrome’ was first identified in Victoria in 1998.
The condition causes development defects within the calves, which are typically born alive, with most surviving to maturity. CA calves appear visibly abnormal with restricted movement caused by reduced elasticity of the connective tissue. Calves will display restricted movement within their front legs, their hind legs will be set back behind them (hyperextension) and will display an upward arching of their spine.
The visible symptoms of the condition become increasingly difficult to detect as the animal grows and the ability for these features to improve over time, is an important characteristic of diagnosing the condition.
CA is controlled by a simple recessive mutation.
Further readingAmerican Angus Association NSW DPI